Purpose: To analyze the fluorescein (FA) and indocyanine green (ICGA) angiographic aspects of serpiginous choroidopathy. Methods: We performed FA and ICGA in 11 consec- utive patients with serpiginous choroidopathy with a SLO (Rodenstock, Germany) and with a still-frame digitized fundus camera (Topcon 50 IA, Japan). Results: In 6 patients the dis- ease was active (12 eyes), in other 5 (10 eyes) it was inactive and 2 of these patients (3 eyes) had developed a choroidal neovascularization. The active lesions were hypofluorescent in the early phases of the FA showing diffusion in the late phases. In 2 of the patients with active disease a retinal vasculitis was also present. The healed lesions were hypofluorescent in the early phase of the FA due to extensive atrophy of the choriocapillaris and choroidal vessels with sparing of major collectors. A progressive hyperfluorescence of the margins was also apparent. In ICGA in the active stage the lesions were hypofluorescent throughout the examination. In 4 of the 6 patients with active disease the ICGA revealed a delayed cho roidal filling next to the active lesions. Follow up showed in only one subject a coincidence between the delayed choroidal filling and the progression of the lesions. Conclusions: FA and ICGA are useful to highlight the level of activity of the disease and the extent of the cho- roidal damage but it cannot precisely predict the evolution of the lesions. CR: None

Fluorescein and indocyanine green angiographic findings in serpiginous choroidopothy.

PIERMAROCCHI, STEFANO;PILOTTO, ELISABETTA;
2000

Abstract

Purpose: To analyze the fluorescein (FA) and indocyanine green (ICGA) angiographic aspects of serpiginous choroidopathy. Methods: We performed FA and ICGA in 11 consec- utive patients with serpiginous choroidopathy with a SLO (Rodenstock, Germany) and with a still-frame digitized fundus camera (Topcon 50 IA, Japan). Results: In 6 patients the dis- ease was active (12 eyes), in other 5 (10 eyes) it was inactive and 2 of these patients (3 eyes) had developed a choroidal neovascularization. The active lesions were hypofluorescent in the early phases of the FA showing diffusion in the late phases. In 2 of the patients with active disease a retinal vasculitis was also present. The healed lesions were hypofluorescent in the early phase of the FA due to extensive atrophy of the choriocapillaris and choroidal vessels with sparing of major collectors. A progressive hyperfluorescence of the margins was also apparent. In ICGA in the active stage the lesions were hypofluorescent throughout the examination. In 4 of the 6 patients with active disease the ICGA revealed a delayed cho roidal filling next to the active lesions. Follow up showed in only one subject a coincidence between the delayed choroidal filling and the progression of the lesions. Conclusions: FA and ICGA are useful to highlight the level of activity of the disease and the extent of the cho- roidal damage but it cannot precisely predict the evolution of the lesions. CR: None
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Utilizza questo identificativo per citare o creare un link a questo documento: https://hdl.handle.net/11577/1480934
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