statistiche ricercatore

Nome	#
null	171
A novel deletion in the GTPase domain of OPA1 causes defects in mitochondrial morphology and distribution, but not in function.	160
Increased mitophagy in the skeletal muscle of spinal and bulbar muscular atrophy patients	151
Reliable and versatile immortal muscle cell models from healthy and myotonic dystrophy type 1 primary human myoblasts	112
Mitochondrial quality control: Cell-type-dependent responses to pathological mutant mitochondrial DNA.	103
Dominant optic atrophy caused by a novel OPA1 mutation: Disruption of the mitochondrial network with preserved bioenergetics.	100
Normal myogenesis and increased apoptosis in myotonic dystrophy type-1 muscle cells.	95
MBNL142 and MBNL143 gene isoforms, overexpressed in DM1-patient muscle, encode for nuclear proteins interacting with Src family kinases	93
INHIBITION OF MITOCHONDRIAL FISSION FAVOURS MUTANT OVER WILD-TYPE MITOCHONDRIAL DNA	91
Mitochondrial DNA heteroplasmy in muscle cybrids harbouring A3243G Melas mutation	49
Functional and molecular responses to stress conditions in heteroplasmic MELAS 3243 RD cybrids.	48
Functional and molecular responses to stress conditions in heteroplasmic MELAS (3243) RD cybrids.	34
Beta-agonist stimulation ameliorates the phenotype of spinal and bulbar muscular atrophy mice and patient-derived myotubes	25
Totale	1.232

	Totale	Lug	Ago	Sett	Ott	Nov	Dic	Gen	Feb	Mar	Apr	Mag	Giu
2018/2019	88	0	0	0	0	0	0	0	0	0	0	47	41
2019/2020	303	33	14	15	15	26	19	37	30	24	58	22	10
2020/2021	245	3	64	20	15	23	6	2	18	27	21	24	22
2021/2022	166	1	6	35	10	17	11	1	23	13	2	14	33
2022/2023	167	23	24	7	29	23	27	0	7	19	0	8	0
2023/2024	107	3	12	14	5	13	25	6	17	8	3	1	0
Totale													1.232