Thus far, there are conflicting results on the causal role of K+ channels in the pathogenesis of acute Oxaliplatin-Induced Peripheral Neurotoxicity (OXAIPN). As such, we tested the hypothesis that the voltage-gated K+ channel KCNN3 repeat polymorphism confers liability to acute OXAIPN.
Liability Of The Voltage-Gated Potassium Channel KCNN3 Repeat Polymorphism To Acute Oxaliplatin-Induced Peripheral Neurotoxicity
Briani, Chiara;
2019
Abstract
Thus far, there are conflicting results on the causal role of K+ channels in the pathogenesis of acute Oxaliplatin-Induced Peripheral Neurotoxicity (OXAIPN). As such, we tested the hypothesis that the voltage-gated K+ channel KCNN3 repeat polymorphism confers liability to acute OXAIPN.File in questo prodotto:
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